High Intraocular Pressure-induced Delayed Diffuse Lamellar Keratitis after Laser In Situ Keratomileusis (LASIK). |
Jin Choi, Won Ryang Wee, Jin Hak Lee, Mee Kum Kim |
1Department of Ophthalmology, Seoul National University College of Medicine, Seoul, Kerea. kmk9@snu.ac.kr 2Seoul Artificial Eye Center, Seoul National University Hospital Clinical Research Institute, Seoul, Korea. 3Department of Ophthalmology, Seoul National University Bundang Hospital, Seongnam, Korea. |
레이저 각막절삭가공성형술 후 안압 상승에 의한 지연성 미만성 층판각막염 1예 |
최진1,2,위원량1,2,이진학2,3,김미금1,2 |
Department of Ophthalmology, Seoul National University College of Medicine1, Seoul, Kerea Seoul Artificial Eye Center, Seoul National University Hospital Clinical Research Institute2, Seoul, Korea Department of Ophthalmology, Seoul National University Bundang Hospital3, Seongnam, Korea |
Correspondence:
Jin Choi, M.D.1,2 |
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Abstract |
PURPOSE To report a case of post-laser in situ keratomileusis (LASIK) delayed diffuse lamellar keratitis (DLK) associated with elevated intraocular pressure (IOP). METHODS: A retrospective review of the medical record for an eye with delayed-onset DLK. RESULTS: In an eye treated with topical steroid eye drops after LASIK, DLK suddenly developed at 6 weeks postoperatively. No corneal epithelial defect was found, but IOP was observed to be high. Keratitis regressed as the IOP was controlled with topical antiglaucoma medications. However, DLK recurred at 3 months postoperatively with increasing IOP again caused by continued topical steroid application. Flap irrigation was performed. No microorganism was found on the interface. After discontinuation of topical steroid, IOP was lowered and DLK disappeared completely. CONCLUSIONS: Elevated IOP can be a causative factor of late-onset DLK after LASIK. |
Key Words:
DLK;IOP;LASIK;Topical steroid |
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